Abstract
Exposure to hyperoxia, invasive mechanical ventilation, and systemic/local sepsis are important antecedents of postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia (BPD). This review will summarize information obtained from animal (baboon, lamb/sheep, rat and mouse) models that pertain to the specific inflammatory agents and signaling molecules that predispose a premature infant to BPD.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 189-201 |
| Number of pages | 13 |
| Journal | Birth Defects Research Part A - Clinical and Molecular Teratology |
| Volume | 100 |
| Issue number | 3 |
| DOIs | |
| State | Published - Mar 2014 |
| Externally published | Yes |
All Science Journal Classification (ASJC) codes
- Pediatrics, Perinatology, and Child Health
- Embryology
- Developmental Biology
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