Projects per year
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Dive into the research topics where James Holaska is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Projects
- 2 Finished
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Emerin regulation of nuclear architecture during cell invasion and metastasi
12/1/19 → 11/29/20
Project: Other project
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Emerin regulation of nuclear architecture during cell invasion and metastasis
12/1/18 → 11/29/19
Project: Other project
Research output
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EDMD-Causing Emerin Mutant Myogenic Progenitors Exhibit Impaired Differentiation Using Similar Mechanisms
Iyer, A. & Holaska, J. M., Jun 15 2020, In: Cells. 9, 6Research output: Contribution to journal › Article › peer-review
Open Access -
Histone acetyltransferase inhibition rescues differentiation of emerin-deficient myogenic progenitors
Bossone, K. A., Ellis, J. A. & Holaska, J. M., Jul 1 2020, In: Muscle and Nerve. 62, 1, p. 128-136 9 p.Research output: Contribution to journal › Article › peer-review
1 Scopus citations -
The molecular basis and biologic significance of the β-dystroglycan-emerin interaction
Gómez-Monsivais, W. L., Monterrubio-Ledezma, F., Huerta-Cantillo, J., Mondragon-Gonzalez, R., Alamillo-Iniesta, A., García-Aguirre, I., Azuara-Medina, P. M., Arguello-García, R., Rivera-Monroy, J. E., Holaska, J. M., Hernández-Méndez, J. M. E., Garrido, E., Magaña, J. J., Winder, S. J., Brancaccio, A., Martínez-Vieyra, I., Navarro-Garcia, F. & Cisneros, B., Sep 1 2020, In: International Journal of Molecular Sciences. 21, 17, p. 1-20 20 p., 5944.Research output: Contribution to journal › Article › peer-review
Open Access -
MAPK signaling pathways and HDAC3 activity are disrupted during differentiation of emerin-null myogenic progenitor cells
Collins, C. M., Ellis, J. A. & Holaska, J. M., Apr 1 2017, In: DMM Disease Models and Mechanisms. 10, 4, p. 385-397 13 p.Research output: Contribution to journal › Article › peer-review
6 Scopus citations -
Diseases of the nucleoskeleton
Holaska, J. M., Oct 1 2016, In: Comprehensive Physiology. 6, 4, p. 1655-1674 20 p.Research output: Contribution to journal › Article › peer-review
3 Scopus citations